A patient at 34 weeks with preeclampsia with severe features is receiving magnesium sulfate at 2 g/hour. Her respiratory rate is 9/min, patellar reflexes are absent, and urine output was 60 mL over the last 4 hours. What is the nurse's priority action?
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Answer: B
Increase the infusion to 3 g/hour to prevent an eclamptic seizure: The findings indicate magnesium toxicity; increasing the dose could cause respiratory or cardiac arrest.
Stop the infusion and prepare calcium gluconate as ordered: Correct. Absent reflexes and respiratory depression signal toxicity; stop magnesium, support breathing, give calcium gluconate per order and check a serum level.
Recheck reflexes in one hour and continue the current rate: Waiting with ongoing infusion allows toxicity to worsen, especially with low urine output reducing excretion.
Give a 500 mL fluid bolus to increase urine output: Fluid boluses risk pulmonary edema in preeclampsia and do not reverse toxicity.
Magnesium toxicity sequence: loss of reflexes, then respiratory depression, then cardiac arrest. Oliguria raises levels because magnesium is renally cleared; the antidote is calcium gluconate.